2. Does caries travel in the direction of the enamel rods because it follows the less mineralised interrod matrix as this is less resistant?
3. What are the components and function of the crevicular fluid?
4. What is the histological composition of pulp?
5. What effect does recession have on dentine?
Does this questions just refer to exposed tubules, stimulus causes hydrodynamic response and this is communicated to the pulp, which sends this message to the brain, which is then translated as pain?
I didn't know where to put these questions so I just started a new post, hope that is okay! :)
I found your post Vanessa.
ReplyDeleteThese are good questions for discussion. Lets see what others have to say.
Sophie
1. Perikymata are grooves along the enamel surface (buccal for example) where the enamel rods and stria of retzius meet. Mamelons are developmental occurances that occur because the are formed from multiple (commonly 3) lobes.
ReplyDelete2. I think so it travels down the interrods because they are less mineralised, therefore needing less demineralisation to allow the caries to travel towards the dentine. The enamel rods are parallel to the interrods, as the interrods are placed between the enamel rods.
4. Pulp is a connective tissue therefore, it
- is vascular
- has a nerve supply
- contains cells
- contains fibers
5. Recession causes dentine to be exposed, and thus exposing the dentinal tubules. Through the hydrodynamic theory any stimulus in the mouth, for example cold or hot water, will cause the osmotic pressure within the tubules to change, forcing the fluid (water) towards the odontoblast cell body at the pulp wall. This is recognised by the pulp and a message or stimulus is sent to the brain and registered as pain. Therefore, recession causes sensitivity to be recognised by dentine.
Recession also causes dentine to become prone to staining and root caries.
Root caries because dentine is less mineralised, therefore allowing the caries to travel faster through the tissue.
Staining as the tissue is less mineralised but also rougher, therefore causing stains to stick to the tissue stronger than it would to enamel (a smooth tissue)
I’m not very sure about question 3 but I hope this helps :)
Thanks Verionica! That helped a lot! :D
ReplyDeleteComponents of the crivecular fluid is mainly WBC's (white blood cells) in particular PMN's( polymorphonucular leukocytes) (think there is also some antigen cells in there not sure if there important or not) these protect tissue from microorganisms and toxins fom the tooth surface and dental biofilm. They do this by traveling through the large interacellular space in the junctional epithelial cells. When there is an increased bacteria load the crivecular fluid componets also increases and the amount.
ReplyDeleteJust read also contains immunological components of blood and sticky plasma protien ??
ReplyDeleteThanks for that Cassie! I found some more helpful information:
ReplyDeleteFunctions
protective effects
clearance of cells and potentially dangerous bacterial molecules;
antibacterial action of immunoglobulins;
(plaque formation induced by calcium ions on the gingival margin)
negative effects
tartar formation induced by alcaline phosphatase;
proteolytic enzyme are dangerous for the gingival sulcus and the other gingival tissues
The production of gingival crevicular fluid seems to be associated with inflammatory processes and not a product of a healty tissue.
Some of its components are probably more involved than others in the developement of the periodontal desease:
cathepsin-K
interleukin-4 and interferon-gamma
leptin
osteocalcin
alcaline phosphatase
http://flipper.diff.org/app/items/info/379